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Models

About 42,523 results
マウス

B2m-KO

系統名:C57BL/6Smoc-B2mem3Smoc
維持形態:Embryo cryopreservation | SMOC番号:NM-KO-18021

Exon2-3 of B2m gene were deleted to generate Ifngr1 knockout mice.

マウス

Tmem119-2A-tdTomato-2A-DTR

系統名:C57BL/6Smoc-Tmem119em1(2A-tdTomato-2A-DTR)Smoc
維持形態:Repository Live | SMOC番号:NM-KI-210117

2A-tdTomato-2A-DTR expression cassette was knocked into the Tmem119 gene.

マウス

Epcam-2A-CreERT2

系統名:C57BL/6Smoc-Epcamem1(2A-CreERT2)Smoc
維持形態:Embryo cryopreservation | SMOC番号:NM-KI-00084

An 2A-CreERT2 co-expression cassette is inserted before 3'UTR region of Epcam gene .

マウス

Adh7-KO

系統名:C57BL/6Smoc-Adh7em1Smoc
維持形態:Embryo cryopreservation | SMOC番号:NM-KO-201121

Exon 3 of Adh7 gene was deleted to generate Adh7 knockout mice.

マウス

hB2M-HLA A2.1(NMG)

系統名:NOD-PrkdcscidIl2rgem1H2-D1em1(hB2M-HLA A2.1)Smoc
維持形態:Developing | SMOC番号:NM-HU-225089

The endogenous mouse H2-D1 gene was replaced by human B2M-HLA A2.1 gene.

マウス

hCD24

系統名:C57BL/6Smoc-Cd24atm1(hCD24)/Smoc
維持形態:Repository Live | SMOC番号:NM-HU-2000060

The endogenous mouse Cd24a gene was replaced by human CD24 gene. While hCD24(2)(Stock No.NM-HU-215025) mice function similarly to hCD24 mice, for more detailed information please contact our technical advisor.

マウス

hCD20

系統名:C57BL/6Smoc-Ms4a1em2(hCD20)Smoc
維持形態:Embryo cryopreservation | SMOC番号:NM-HU-190068

The endogenous mouse Cd20(Ms4a1) gene was replaced by human CD20 gene. While hCD20(2)(Stock No.NM-HU-200114) mice function similarly to hCD20 mice,for more detailed information please contact our technical advisor.

マウス

hTLR9

系統名:C57BL/6Smoc-Tlr9tm1(hTLR9)Smoc
維持形態:Embryo cryopreservation | SMOC番号:NM-HU-18016

This TLR9 humanized mouse model was established by replacing the mouse Tlr9 gene exon2 with the human TLR9 corresponding sequence by homologous recombination.

マウス

F9-KO

系統名:C57BL/6Smoc-F9em1Smoc
維持形態:Embryo cryopreservation | SMOC番号:NM-KO-18046

The F9 gene is located on the X chromosome. In this F9 knockout mouse model, gRNAs were designed targeting Exon8 of F9 gene via CRISPR gene editing technology. Loss of F9 caused coagulopathy in mice. Mice that are homozygous for F9 knockout are viable, fertile and normal in size. Normal fighting in the cage may cause bleeding or even death due to massive internal hemorrhaging. After the tail cutting, wounds must be cauterized to prevent homozygous knockout mice from blood loss and death. This strain is a powerful model for studying coagulopathy, gene therapy methods and function of factor IX mutations. F9-KO mice (Stock No.NM-KO-18046)carry a knockout allele derived from the targeted deletion of exon 8. While F9-KO(2) mice (Stock No.NM-KO-200607) carrying the exon 1-8 deletion.

マウス

hTNFR2

系統名:C57BL/6Smoc-Tnfrsf1bem2(hTNFRSF1B)Smoc
維持形態:Embryo cryopreservation | SMOC番号:NM-HU-190010

The endogenous mouse Tnfrsf1b gene was replaced by human TNFRSF1B gene.

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